The carrier proteins through which sugar enters pancreatic beta cells have "Higher Km" (a high Km indicates low affinity for the substrate) than the carriers through which sugar enters muscle cells at fasting blood sugar levels.
In order for muscle cells to increase sugar import Insulin "must be present". Oh, and glucokinase has a High Km (a lower affinity for glucose ) when compared to hexokinase.
"Proton pumps" are responsible for the release of transferrin from the transferrin receptor and iron form transferrin (Proton pumps are transporters that move hydrogen ions from regions with low concentration to regions with higher concentration)
"Iron Reductases" convert the iron to the active form in which it may be exported (ferric iron is converted to ferrous iron by ferric reductase). This happens in the "Lysosome" (lysosome is an organelle that contains hydrolytic enzymes)